The blood-spinal twine barrier (BSCB) performs a key function in sustaining the microenvironment and is primarily composed of tight junction proteins and nonfenestrated capillary endothelial cells. After damage, BSCB injury ends in growing capillary permeability and launch of inflammatory elements. Latest research have reported that haem oxygenase-1 (HO-1) fragments missing 23 amino acids on the […]
![Adenovirus-delivered GFP-HO-1C[INCREMENT]23 attenuates blood-spinal cord barrier permeability after rat spinal cord contusion.](https://www.korilog.com/drh/uploads/2021/10/Capture-2.png)